The Fundamentals On Mitochondrial Vitamins

By Tracie Knight


The global health research shows that most people do not have sufficient intake and access to Vitamin D. Deficiency in affected individuals are characterized with rickets formation and muscle strains. The mitochondrial vitamins C and D are fundamental remedy for such patients. The vitamin D is gotten mostly from sun energy. It aids in bone formation thus caretakers of young children are always advised to expose the infant to sun energy at given moments. Foods such as fish liver oil and fish are also sources.

Vitamin D enhances workability of mitochondria and bone formation and its deficiency leads to formation of rickets and muscle fatigue due to reduced efficiency on functioning of mitochondria. This organelle acts as power station of cell organelle that provides energy for all activities on cell and it makes this energy by use of oxygen and glucose.

Adenosine Triphosphate is a form of energy that exists in cell organelle. The hormone phosphocreatine aids in production of Adenosine Triphosphate in muscles. Mitochondrion helps in rejuvenation of phosphocreatine as the muscles contract. Low levels affect the functioning and production of this important nutrient.

Recent researches are on the look for health issues that are emerging based on its role in human body. They have far considered a test on patients, where a dosage was given orally for a period of ten to twelve weeks. It was noted that the supplements in the oral dosage given, led to a decrease in the functioning of the mitochondria and time recovery on the phosphocreatine.

Incorporation on dual relationship between Vitamin C and mitochondria are being investigated further. Its importance helps reduce deficiency causes such as gum disease and hair loss, which is most likely in aging individuals. The GLUT10 is a glucose that transports oxidized vitamin c in the mitochondrial cell. It aid on protection of cell against oxidative stress.

Its detection to its existence on unit organelle is being used to understand on inherited causes of arterial disorder tortousity disease of the connective tissues. Oxidative stress is due to Cell imbalances of reactive chemical molecules, this unevenness hold the capacity of cell organelle to detoxicate it and fix dilapidate tissues and on reactive oxygen species.

The mitochondria have been predicted to be the production site of reactive oxygen species. Recycling of the dehydroascorbic acid also benefits from its activity. The connection on molecular mechanism based on dehydroascorbic acid is still a puzzle to be unraveled. Some cases of arterial tortousity syndrome are due to abnormalities in the GLUT10. This quasi finding has been applied on comprehension of these type 2 diabetes and GLUT10 as a reinforcement of ROS and DHA.

Human body require nutrients to function properly, the need to take a diet meal will help in body fight pathogens on its own natural mechanism. Improving your immunity is also of much use since administration of drugs is not that efficient. Then if this is so then why waiting till you contract the disease yet you have the preventive measures at hand. These measures range from massive intake of fruits and vegetables on a diet basis.




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